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Is Immunostaining for Helicobacter pylori H. pylori

Is Immunostaining for Helicobacter pylori H. pylori Applied Immunohistochemistry & Molecular Morphology 10(1): 82–84, 2002 © 2002 Lippincott Williams & Wilkins, Inc., Philadelphia Is Immunostaining for Helicobacter pylori Superior to the Special Stain Thiazine in Detecting Small Numbers of H. pylori in Gastric Biopsies? Nirag Jhala, M.D., Sarah Lechago, B.S., Juan Lechago, M.D., Ph.D., and Mamoun Younes, M.D. Helicobacter pylori is implicated in the pathogenesis of gastri- cers (1,2) and active gastritis, and leads to regression of tis and duodenal ulcers, gastric lymphoma of the mucosa- early stage mucosa-associated lymphoid tissue (MALT) associated lymphoid tissue (MALT) type, and gastric adeno- lymphoma (3,4). Thus, patients with documented H. py- carcinoma. Eradication of H. pylori with antibiotic therapy lori infection are treated aggressively with a combination therefore is essential, not only for the successful treatment of of drugs that include proton pump inhibitors and antibi- active gastritis, but also for the treatment and prevention of the MALT lymphoma. It has been suggested recently that immu- otics (2,5,6). Documentation of the complete absence of nostaining for H. pylori is more sensitive than special stains for H. pylori after anti–H. pylori therapy, therefore, is of the detection of the organism in the gastric biopsies after triple paramount importance for the http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Applied Immunohistochemistry & Molecular Morphology Wolters Kluwer Health

Is Immunostaining for Helicobacter pylori H. pylori

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1062-3345
eISSN
1533-4058

Abstract

Applied Immunohistochemistry & Molecular Morphology 10(1): 82–84, 2002 © 2002 Lippincott Williams & Wilkins, Inc., Philadelphia Is Immunostaining for Helicobacter pylori Superior to the Special Stain Thiazine in Detecting Small Numbers of H. pylori in Gastric Biopsies? Nirag Jhala, M.D., Sarah Lechago, B.S., Juan Lechago, M.D., Ph.D., and Mamoun Younes, M.D. Helicobacter pylori is implicated in the pathogenesis of gastri- cers (1,2) and active gastritis, and leads to regression of tis and duodenal ulcers, gastric lymphoma of the mucosa- early stage mucosa-associated lymphoid tissue (MALT) associated lymphoid tissue (MALT) type, and gastric adeno- lymphoma (3,4). Thus, patients with documented H. py- carcinoma. Eradication of H. pylori with antibiotic therapy lori infection are treated aggressively with a combination therefore is essential, not only for the successful treatment of of drugs that include proton pump inhibitors and antibi- active gastritis, but also for the treatment and prevention of the MALT lymphoma. It has been suggested recently that immu- otics (2,5,6). Documentation of the complete absence of nostaining for H. pylori is more sensitive than special stains for H. pylori after anti–H. pylori therapy, therefore, is of the detection of the organism in the gastric biopsies after triple paramount importance for the

Journal

Applied Immunohistochemistry & Molecular Morphology – Wolters Kluwer Health

Published: Mar 1, 2002

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