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Fluorescent In-situ Hybridization Study of Non-papillary Oncocytic/Eosinophilic Renal Cell Carcinoma

Fluorescent In-situ Hybridization Study of Non-papillary Oncocytic/Eosinophilic Renal Cell... CASE REPORT Fluorescent In-situ Hybridization Study of Non-papillary Oncocytic/Eosinophilic Renal Cell Carcinoma Kien T. Mai, MD,*w Itrat Ahmed, MD,*w Joelle Levac, MLT,*w and Bich N. Nguyen, MD*w cytoplasm and are associated with loss of chromosome Aims: Papillary renal cell carcinoma (PRCC) and clear cell RCC 3p. The small arm 3p is known to harbor tumor (CRCC) can display extensive areas with oncocytic/eosinophilic suppressor (recessive) genes Von Hippel Lindau (VHL) changes and may be associated with either minimal papillary and fragile histidine triad (FHIT) at loci p25 or 14, 1–12 architecture. These nonpapillary oncocytic/eosinophilic RCC respectively. PRCC consists of chromophil cells often mimic renal oncocytoma (RO). We investigated numeric forming papillary structures and are characterized by changes of chromosomes 7, 17, and Y and loss of the small arm specific cytogenetic alterations with trisomy 7/17 and loss 13–16 of chromosome 3 in the above-mentioned oncocytic RCC by of chromosome Y. CRCC may be associated with using the florescent in-situ hybridization (FISH). extensive areas of eosinophilic cytoplasm and PRCC may display a solid cell growth pattern and oncocytic changes. Materials and Methods: Archival cases of oncocytic RCC The eosinophilic variant of CRCC and the oncocytic and previously screened by immunohistochemical study http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Applied Immunohistochemistry & Molecular Morphology Wolters Kluwer Health

Fluorescent In-situ Hybridization Study of Non-papillary Oncocytic/Eosinophilic Renal Cell Carcinoma

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Copyright
Copyright #x00A9; 2010 by Lippincott Williams & Wilkins
ISSN
1541-2016
DOI
10.1097/PAI.0b013e3181ec51a9
pmid
20861792
Publisher site
See Article on Publisher Site

Abstract

CASE REPORT Fluorescent In-situ Hybridization Study of Non-papillary Oncocytic/Eosinophilic Renal Cell Carcinoma Kien T. Mai, MD,*w Itrat Ahmed, MD,*w Joelle Levac, MLT,*w and Bich N. Nguyen, MD*w cytoplasm and are associated with loss of chromosome Aims: Papillary renal cell carcinoma (PRCC) and clear cell RCC 3p. The small arm 3p is known to harbor tumor (CRCC) can display extensive areas with oncocytic/eosinophilic suppressor (recessive) genes Von Hippel Lindau (VHL) changes and may be associated with either minimal papillary and fragile histidine triad (FHIT) at loci p25 or 14, 1–12 architecture. These nonpapillary oncocytic/eosinophilic RCC respectively. PRCC consists of chromophil cells often mimic renal oncocytoma (RO). We investigated numeric forming papillary structures and are characterized by changes of chromosomes 7, 17, and Y and loss of the small arm specific cytogenetic alterations with trisomy 7/17 and loss 13–16 of chromosome 3 in the above-mentioned oncocytic RCC by of chromosome Y. CRCC may be associated with using the florescent in-situ hybridization (FISH). extensive areas of eosinophilic cytoplasm and PRCC may display a solid cell growth pattern and oncocytic changes. Materials and Methods: Archival cases of oncocytic RCC The eosinophilic variant of CRCC and the oncocytic and previously screened by immunohistochemical study

Journal

Applied Immunohistochemistry & Molecular MorphologyWolters Kluwer Health

Published: Jan 1, 2011

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