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EDITORIAL URRENT HIV and cardiovascular disease: recognizing and PINION reducing disparities a,b,c Virginia A. Triant INTRODUCTION determinants of health in relation to CVD risk in PWH is only beginning to be elucidated. Conse- Advances in antiretroviral therapy (ART) have quently, CVD in PWH is thought to be the result resulted in increased longevity for people with of a complex interplay of multiple factors and path- HIV (PWH). Yet, this aging HIV population is con- ways, with HIV itself and HIV-specific factors acting fronting a disproportionately increasing burden of as risk-enhancers [5]. Current strategies for risk- chronic noncommunicable diseases (NCDs) com- stratification, prevention, and management of pared with people without HIV [1]. Salient among CVD in PWH, however, are transported from the these chronic diseases that represent barriers to general population and do not reflect the unique healthy aging is cardiovascular disease (CVD) [2– factors – including sociodemographic and immu- 4]. Extensive research over the past decades indi- nologic – driving excess CVD risk in PWH [5,9,10]. cates that HIV-associated CVD is more prevalent, is Articles in this issue address current knowledge mechanistically distinct, and is undertreated com- and knowledge gaps across the spectrum of cardio- pared with CVD in
Current Opinion in HIV and AIDS – Wolters Kluwer Health
Published: Sep 1, 2022
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