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J. Chatterjea, S. Swarup, S. Gosh (1961)
Primaquine SensitivityBritish Medical Journal, 2
R. Gross, R. Hurwitz, P. Marks (1958)
An hereditary enzymatic defect in erythrocyte metabolism: glucose-6-phosphage dehydrogenase deficiency.The Journal of clinical investigation, 37 8
W. Zinkham, R. Lenhard, B. Childs (1958)
A deficiency of glucose-6-phosphate dehydrogenase activity in erythrocytes from patients with favism.Bulletin of the Johns Hopkins Hospital, 102 4
P. Carson, C. Flanagan, Ickes Ce, A. Alving (1956)
Enzymatic deficiency in primaquine-sensitive erythrocytes.Science, 124 3220
E. Beutler (1957)
The glutathione instability of drug-sensitive red cells; a new method for the in vitro detection of drug sensitivity.The Journal of laboratory and clinical medicine, 49 1
Tarlov Tarlov, Brewer Brewer, Carson Carson, Alvtng Alvtng (1962)
Primaquine sensitivityArch. Int. Med., 109
A. Adam (1961)
Linkage between Deficiency of Glucose-6-phosphate Dehydrogenase and Colour-blindnessNature, 189
Szeinberg Szeinberg, Sheba Sheba, Adam Adam, Ramot Ramot (1959)
An hereditary abnormality of the metabolism of glutathione in red blood cellsVllth Congr. Genet. Haematologicae, Borne,, 8
(1960)
Genetice. Indagini genetiche sulla predisposizione a1 favismo : 11. Dati familiari. Associazione genica con il daltonismo
Childs Childs, Zinkham Zinkham, Browne Browne, Kimbro Kimbro, Torbert Torbert (1958)
A genetic study of a defect in glutathione metabolism of the erythrocyteBull. Johns Hopkins Hosp., 102
M. Siniscalco, L. Bernini, B. Latte, A. Motulsky (1961)
Favism and Thalassæmia in Sardinia and their Relationship to MalariaNature, 190
P. Marks, J. Banks, R. Gross (1962)
Genetic Heterogeneity of Glucose-6-Phosphate Dehydrogenase DeficiencyNature, 194
(1960)
Genetice
(1961)
Favism and thal&saemia in Sardinia
Beutler Beutler, Yeh Yeh, Fairbanks Fairbanks (1962)
The normal human female as a mosaic of X‐chromosome activity: studies using the gene for g‐6‐pd‐deficiency as a markerProc. Nat. Acad. Sci., Wash.,, 48
P. Marks (1958)
Red cell glucose-6-phosphate and 6-phosphogluconic dehydrogenases and nucleoside phosphorylase.Science, 127 3310
A. Tarlov, G. Brewer, P. Carson, A. Alving (1962)
Primaquine sensitivity. Glucose-6-phosphate dehydrogenase deficiency: an inborn error of metabolism of medical and biological significance.Archives of internal medicine, 109
A. Adam, C. Sheba, R. Race, R. Sanger, P. Tippett, J. Hamper, J. Gavin (1962)
LINKAGE RELATIONS OF THE X-BORNE GENES RESPONSIBLE FOR GLUCOSE-6-PHOSPHATE DEHYDROGENASE AND FOR THE Xg BLOOD-GROUPSThe Lancet, 279
(1958)
Red cell glucose-6-phosphate and 6-phospho~luconic
A. Alving, R. Kellermeyer, A. Tarlov, S. Schrier, P. Carson (1958)
Biochemical and genetic aspects of primaquine-sensitive hemolytic anemia.Annals of internal medicine, 49 2
I. Porter, J. Schulze, V. McKusick (1962)
Linkage between Glucose-6-Phosphate Dehydrogenase Deficiency and Colour-BlindnessNature, 193
W. Park (1957)
The occurrence of sex chromatin in early human and macaque embryos.Journal of anatomy, 91 3
Beutleb Beutleb, Robson Robson, Buttenweiser Buttenweiser (1957)
The glutathione instability of drug‐sensitive red cellsJ. Lab. Clin. Med., 49
(1962)
Genetic implications of DFP 3 * - erythrocyte survival studies in negro females heterozygous for glucose - 6 - phosphate dehydrogenase deficiency
(1960)
Genetice . Indagini genetiche sulla predisposizione a 1 favismo : 11 . Dati familiari . Associazione genica con il daltonismo . Accad
(1958)
An hereditary enzymatic defect in erythrocyte LYON
(1960)
Drug - induced hemolytic anemia ( primaquine sensitivity )
A. Szeinberg, C. Sheba, A. Adam, B. Ramot (1959)
A hereditary abnormality of the metabolism of glutathione in the red blood cells.Acta geneticae medicae et gemellologiae, 8
J. Stewart (1961)
GENETIC FACTORS ON THE X CHROMOSOMEThe Lancet, 278
E. Beutler, M. Yeh, V. Fairbanks (1962)
THE NORMAL HUMAN FEMALE AS A MOSAIC OF X-CHROMOSOME ACTIVITY: STUDIES USING THE GENE FOR G-6-PD-DEFICIENCY AS A MARKERProceedings of the National Academy of Sciences of the United States of America, 48
SUMMARY 1 Various glucose‐6‐phosphate dehydrogenase‐deficient pedigrees from the literature violating classical patterns of sex‐linked inheritance are reviewed and discussed. 2 It is suggested that anomalous phenotypes observed in these families may be the result of epistatic reactions between the effects of abnormal sex‐linked genes or gene and an autosomal pair.
Annals of Human Genetics – Wiley
Published: Mar 1, 1963
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