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P. Nelson (1959)
Human Chromosomal AbnormalitiesBritish Medical Journal, 2
L. Penrose (1934)
The Relative Aetiological Importance of Birth Order and Maternal Age in MongolismProceedings of The Royal Society B: Biological Sciences, 115
Lenz Lenz (1959b)
Der Einfluss des Alters der Eltern und Geburtennummer auf angeborene pathologische Zustande beim Kind. II. Spezieller TeilActa genet., 9
J. Stewart (1960)
Mechanisms of Meiotic Non-disjunction in ManNature, 187
W. Lenz (1959)
DER EINFLUSS DES ALTERS DER ELTERN UND DER GEBURTENNUMMER AUF ANGEBORENE PATHOLOGISCHE ZUSTÄNDE BEIM KINDHuman Heredity, 9
A. Johnston, M. Ferguson-Smith, S. Handmaker, H. Jones, G. Jones (1961)
The triple-X syndrome. Clinical, pathological, and chromosomal studies in three mentally retarded cases.British medical journal, 2 5259
Lenz Lenz (1959a)
Der Einfluss des Alters der Eltern und der Geburtennummer auf angeborene pathologische Zustände beim Kind. I. Allgemeine methodische GesichtspunkteActa genet., 9
Penrose Penrose (1934)
The relative aetiological importance of birth order and maternal age in mongolismProc. Boy. Soc. B, 115
David Smith, K. Patau, E. Therman, S. Inhorn (1960)
A new autosomal trisomy syndrome: multiple congenital anomalies caused by an extra chromosome.The Journal of pediatrics, 57
A. Johnston, M. Ferguson-Smith, S. Handmaker, H. Jones, G. Jones (1961)
The Triple-X SyndromeBritish Medical Journal, 2
M. Ferguson-Smith (1960)
Cytogenetics in man.Archives of internal medicine, 105
(1959)
See Davidson & Robertson (1961), p
C. Browning, J. Cohen, S. Ellingworth, R. Gulbransen (1926)
The Antiseptic Properties of the Amino Derivatives of Styryl and Anil QuinolineProceedings of The Royal Society B: Biological Sciences, 100
Summary Among seventy patients with nuclear sex chromatin Turner's syndrome sixty‐three individuals were found who had at least one liveborn sib. In these sixty‐three families the observed maternal age (25.413 ± 5.494, S.E. 0.692) did not differ significantly from the expected maternal mean (26.091 ± 5–262); nor did the paternal observed age (27.742 ± 5.651, S.E. 0.718) differ significantly from the expected paternal mean (28.421 ± 5.612). The observed birth ordinal (2.127 ± 1.550, S.E. 0.195) was not significantly different from expected birth ordinal (2.405 ± 1.644). Limitations due to residual fertility of parents are, in this survey, regarded as minimal. It is a pleasure to acknowledge that the beginnings of this survey lay in conversations between Prof. L. S. Penrose and one of us (S. H. B.). We wish to thank Mrs Barbara Latrobe and Mrs Jane Schultz for their assistance.
Annals of Human Genetics – Wiley
Published: Mar 1, 1962
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