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INTRODUCTIONInflammatory processes are included in all states of atherosclerotic disease progression and are literally important for the initiation of acute coronary syndromes (ACSs). Accordingly, data from randomized controlled trials had reported that intensive lipid lowering with statins leads to additional clinical benefits in the same clinical settings. Statins (competitive inhibitors of 3‐hydroxy‐3‐methylglutaryl coenzyme A reductase) are associated with marked risk reduction in patients with coronary artery disease (CAD). owing to their lipid lowering effect and the pleiotropic characteristics they exhibit, both in vivo and in vitro It has been shown that this class of drugs decreases reactive oxygen species generation, limits smooth muscle cells proliferation, has anti‐thrombotic and anti‐inflammatory effects, augments the production of nitric oxide in endothelial cells and stabilizes vulnerable atherosclerotic plaques. Additional non‐lipid dependent merits include inhibition of neurohormonal mechanisms (eg, renin‐angiotensin system), attenuation of ventricular remodeling (eg, altering matrix metalloproteinase activity and controlling fibrosis), decreasing rate of myocardial necrosis and augmentation of neoangiogenesis. These different mechanisms of action contribute to limitation of infarct area size and better left ventricle (LV) systolic function. B‐type natriuretic peptide (BNP) is a neurohormone (32‐amino acids) that is synthesized in the form of pre‐pro BNP, which is cleaved producing pro‐BNP,
Journal of Interventional Cardiology – Wiley
Published: Jan 1, 2017
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