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Prenatal ethanol exposure has been associated with long‐lasting intellectual impairments in children. Previous studies using animal models of fetal ethanol exposure suggest that these deficits are, at least in part, linked to neurochemical abnormalities in the hippocampal formation. We explored whether prenatal exposure to moderate quantities of ethanol produced functional deficits at the entorhinal cortical perforant path‐dentate granule cell connection by examining some electrophysiological properties, including the induction of long‐term potentiation (LTP).
Hippocampus – Wiley
Published: Jan 1, 1997
Keywords: ; ;
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