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(1965)
Ttwria delle frequenze di incompatibilita di trapianto
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General purpose probability models in histocompatibility testing. I âStrong â and âweak â alleles at one locus. BY REGINA C . ELANDT-JOHNSONâ Department of Biostatistics, University of North Carolina, Chapel Hill, North Carolina, U.S.A. I . INTRODUCTION. IMMUNOGENETIC MECHANISM OF HISTOCOMPATIBILITY 1.1. The general model for allotransplantation of tissue is âimmunogenetic â, and its basic concepts of response are very much similar to those in blood transfusion. Where the donor possesses an antigen (or antigens), called âhistocompatibility antigen(s) â, which is lacking in the recipient, antibodies against the donorâs antigen(s) are manufactured (mostly in lymph nodes and spleen) and, after a certain time, the graft is rejected. But if the recipient has at least all the antigens possessed by the donor, the graft should be accepted. The tissue compatibility (histocompatibility) is under genetic control; that is, the histocompatibility antigens are the gene products. We do not attempt here to discuss the possible relations: gene-antigen-antibody, since the terminology is not clearlyestablished. There is someevidence that, for example, the relatively wellknown histocompatibility locus H-2 in mouse is really a H - 2 âregion â (or âcomplex locusâ). Each â site â (or pseudo-allele) controls one antigenic specificity (or determinant) which
Annals of Human Genetics – Wiley
Published: Jan 1, 1968
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