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Diseases of the Cardiovascular System

Diseases of the Cardiovascular System Bv R. J. BING2 The Medical College of A labama, University of A labama, Birmingham, A labama CORONARY HEART DISEASE Physiologic as well as anatomic studies have contributed to an under­ standing of coronary heart disease during the last year. The problem of wheth­ er or not the shock-like syndrome following infarction results from toxic substances or hemodynamic factors has been dealt with by Freis and as­ sociates (1) and by Agress et al. (2). Freis and associates found that myo­ cardial infarction resulted in a reduction of cardiac output and an increase in total peripheral resistance, central venous pressure, and circulation time. They found a slight reduction in total blood volume. Apparently infarction results in a reduction in stroke volume because of the myocardial injury. A series of compensatory mechanisms then follow, which produce a clinical picture resembling shock. Congestive heart failure following myocardial in­ farction is the result of intensive vasocontriction existing in the presence of a reduced stroke volume, resulting in fluid retention. In contrast, Agress and associates found that in artifically produced myocardial infarction in dogs there was a total blood volume loss of per cent. Investigation of coronary blood flow in man using http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Annual Review of Medicine Annual Reviews

Diseases of the Cardiovascular System

Bing, R J
Annual Review of Medicine , Volume 4 (1) – Feb 1, 1953
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Publisher
Annual Reviews
Copyright
Copyright 1953 Annual Reviews. All rights reserved
Subject
Review Articles
ISSN
0066-4219
eISSN
1545-326X
DOI
10.1146/annurev.me.04.020153.000415
pmid
13105267
Publisher site
See Article on Publisher Site

Abstract

Bv R. J. BING2 The Medical College of A labama, University of A labama, Birmingham, A labama CORONARY HEART DISEASE Physiologic as well as anatomic studies have contributed to an under­ standing of coronary heart disease during the last year. The problem of wheth­ er or not the shock-like syndrome following infarction results from toxic substances or hemodynamic factors has been dealt with by Freis and as­ sociates (1) and by Agress et al. (2). Freis and associates found that myo­ cardial infarction resulted in a reduction of cardiac output and an increase in total peripheral resistance, central venous pressure, and circulation time. They found a slight reduction in total blood volume. Apparently infarction results in a reduction in stroke volume because of the myocardial injury. A series of compensatory mechanisms then follow, which produce a clinical picture resembling shock. Congestive heart failure following myocardial in­ farction is the result of intensive vasocontriction existing in the presence of a reduced stroke volume, resulting in fluid retention. In contrast, Agress and associates found that in artifically produced myocardial infarction in dogs there was a total blood volume loss of per cent. Investigation of coronary blood flow in man using

Journal

Annual Review of Medicine – Annual Reviews

Published: Feb 1, 1953

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